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dc.contributorVall d'Hebron Barcelona Hospital Campus
dc.contributor.authorOria, Marc
dc.contributor.authorDuru, Soner
dc.contributor.authorFigueira, Rebeca L
dc.contributor.authorScorletti, Federico
dc.contributor.authorTurner, Lucas E
dc.contributor.authorFernandez-Alonso, Irati
dc.contributor.authorFernández Martín, Alejandra
dc.contributor.authorMarotta Baleriola, Mario
dc.date.accessioned2020-08-04T10:41:25Z
dc.date.available2020-08-04T10:41:25Z
dc.date.issued2019-09-26
dc.identifier.citationOria M, Duru S, Figueira RL, Scorletti F, Turner LE, Fernandez-Alonso I, et al. Cell necrosis, intrinsic apoptosis and senescence contribute to the progression of exencephaly to anencephaly in a mice model of congenital chranioschisis. Cell Death Dis. 2019 Sep 26;10:721.
dc.identifier.issn2041-4889
dc.identifier.urihttps://hdl.handle.net/11351/5134
dc.descriptionLíquid amniòtic; Mortalitat neonatal; Exencefàlia
dc.description.sponsorshipThis work was supported by Prof. Jose L. Peiro internal Cincinnati Children's Hospital funding.
dc.language.isoeng
dc.publisherSpringer Nature
dc.relation.ispartofseriesCell Death & Disease;10
dc.rightsAttribution 4.0 International
dc.rights.urihttp://creativecommons.org/licenses/by/4.0/
dc.sourceScientia
dc.subjectFetus - Cervell - Malformacions
dc.subjectRates (Animals de laboratori)
dc.subjectLíquid amniòtic
dc.subject.meshNervous System Malformations
dc.subject.mesh/mortality
dc.subject.meshMice, Neurologic Mutants
dc.subject.meshAmniotic Fluid
dc.titleCell necrosis, intrinsic apoptosis and senescence contribute to the progression of exencephaly to anencephaly in a mice model of congenital chranioschisis
dc.typeinfo:eu-repo/semantics/article
dc.identifier.doi10.1038/s41419-019-1913-6
dc.subject.decsmalformaciones del sistema nervioso
dc.subject.decs/mortalidad
dc.subject.decsratones mutantes neurológicos
dc.subject.decslíquido amniótico
dc.relation.publishversionhttps://www.nature.com/articles/s41419-019-1913-6
dc.type.versioninfo:eu-repo/semantics/publishedVersion
dc.audienceProfessionals
dc.contributor.authoraffiliation[Oria M, Duru S, Fernandez-Alonso I] Center for Fetal and Placental Research, Cincinnati Children's Hospital Medical Center (CCHMC), Cincinnati, USA. [Figueira RL] Center for Fetal and Placental Research, Cincinnati Children's Hospital Medical Center (CCHMC), Cincinnati, USA. Laboratory of Experimental Fetal Surgery "Michael Harrison", Division of Pediatric Surgery, Department of Surgery and Anatomy, Ribeirao Preto Medical School, University of Sao Paulo-USP, Ribeirao Preto, Brazil. [Scorletti F] Center for Fetal and Placental Research, Cincinnati Children's Hospital Medical Center (CCHMC), Cincinnati, USA. Department of Pediatric Surgery, Hospital Bambino Gesu, Rome, Italy. [Turner LE] The Chicago Institute for Fetal Health, Ann & Robert H. Lurie Children's Hospital of Chicago, Chicago, USA. Department of Pediatric Surgery, Northwestern University, Feinberg School of Medicine, Chicago, USA. [Fernandez-Martin A, Marotta M] Center for Fetal and Placental Research, Cincinnati Children's Hospital Medical Center (CCHMC), Cincinnati, USA. Laboratori de Bioenginyeria, Teràpia Cel•lular i Cirurgia en Malformacions Congènites, Vall d'Hebron Institut de Recerca (VHIR), Barcelona, Spain. Universitat Autònoma de Barcelona, Barcelona, Spain
dc.identifier.pmid31558708
dc.identifier.wos000488854900011
dc.rights.accessrightsinfo:eu-repo/semantics/openAccess


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