dc.contributor | Vall d'Hebron Barcelona Hospital Campus |
dc.contributor.author | Diez, Emiliano Raúl |
dc.contributor.author | Sanchez Garcia, Jose A |
dc.contributor.author | Prado, Natalia Jorgelina |
dc.contributor.author | Ponce Zumino, Amira Zulma |
dc.contributor.author | García-Dorado García, David |
dc.contributor.author | Miguel Miatello, Roberto |
dc.contributor.author | Rodríguez Sinovas, Antonio |
dc.date.accessioned | 2021-04-14T07:51:01Z |
dc.date.available | 2021-04-14T07:51:01Z |
dc.date.issued | 2019-11-25 |
dc.identifier.citation | Diez E, Sánchez JA, Prado NJ, Ponce Zumino AZ, García-Dorado D, Miatello R, et al. Ischemic Postconditioning Reduces Reperfusion Arrhythmias by Adenosine Receptors and Protein Kinase C Activation but Is Independent of KATP Channels or Connexin 43. Int J Mol Sci. 2019 Nov 25;20(23):5927. |
dc.identifier.issn | 1422-0067 |
dc.identifier.uri | https://hdl.handle.net/11351/5848 |
dc.description | Arrítmia; Receptors d’adenosina; Canals KATP |
dc.description.sponsorship | This study was supported by Secretaría de Investigación, Internacionales y Posgrado, Universidad Nacional de Cuyo (06/J505) and by the Spanish, Ministerio de Ciencia, Innovación y Universidades, Instituto de Salud Carlos III (CIBERCV), cofinanced by the European Regional Development Fund (ERDF-FEDER, a way to build Europe), and by Fundació La Marató de TV3 (n°. 201536-10). Antonio Rodríguez-Sinovas has a consolidated Miguel Servet contract. Jose A. Sánchez was supported by the International Research Training Group 1566 on Protecting the Heart from Ischemia (PROMISE). |
dc.language.iso | eng |
dc.publisher | MDPI |
dc.relation.ispartofseries | International Journal of Molecular Sciences;20(23) |
dc.rights | Attribution 4.0 International |
dc.rights.uri | http://creativecommons.org/licenses/by/4.0/ |
dc.source | Scientia |
dc.subject | Arrítmia |
dc.subject | Adenosina |
dc.subject.mesh | Arrhythmias, Cardiac |
dc.subject.mesh | Ischemic Postconditioning |
dc.title | Ischemic Postconditioning Reduces Reperfusion Arrhythmias by Adenosine Receptors and Protein Kinase C Activation but Is Independent of KATP Channels or Connexin 43 |
dc.type | info:eu-repo/semantics/article |
dc.identifier.doi | 10.3390/ijms20235927 |
dc.subject.decs | arritmias cardíacas |
dc.subject.decs | poscondicionamiento isquémico |
dc.relation.publishversion | https://www.mdpi.com/1422-0067/20/23/5927 |
dc.type.version | info:eu-repo/semantics/publishedVersion |
dc.audience | Professionals |
dc.contributor.organismes | Institut Català de la Salut |
dc.contributor.authoraffiliation | [Diez ER, Prado NJ, Ponce Zumino AZ, Miatello RM] Facultad de Ciencias Médicas, Universidad Nacional de Cuyo, Mendoza 5500, Argentina. Institute of Medical and Experimental Biology of Cuyo, CONICET, Mendoza 5500, Argentina. [Sánchez JA, García-Dorado D, Rodríguez-Sinovas A] Grup Malalties Cardiovasculars, Vall d’Hebron Institut de Recerca (VHIR), Barcelona, Spain. Vall d’Hebron Hospital Universitari, Barcelona, Spain. Departament de Medicina, Universitat Autònoma de Barcelona, Bellaterra, Spain |
dc.identifier.pmid | 31775376 |
dc.identifier.wos | 000504428300109 |
dc.rights.accessrights | info:eu-repo/semantics/openAccess |